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dc.contributor.authorGoldgraben, M. A.
dc.contributor.authorFewings, E.
dc.contributor.authorLarionov, A.
dc.contributor.authorScarth, J.
dc.contributor.authorRedman, J.
dc.contributor.authorTelford, Nicholas
dc.contributor.authorArkwright, P. D.
dc.contributor.authorBonney, D.
dc.contributor.authorWilks, Deepti P
dc.contributor.authorKulkarni, Samar
dc.contributor.authorTaylor, A. M. R.
dc.contributor.authorTischkowitz, M. D.
dc.contributor.authorMeyer, Stefan
dc.date.accessioned2020-06-16T11:03:11Z
dc.date.available2020-06-16T11:03:11Z
dc.date.issued2020en
dc.identifier.citationM. A. Goldgraben, E. Fewings, A. Larionov et al. Genomic profiling of acute myeloid leukaemia associated with ataxia telangiectasia identifies a complex karyotype with wild-type TP53 and mutant KRAS, G3BP1 and IL7R. Pediatr Blood Cancer. 2020:e28354en
dc.identifier.pmid32383811en
dc.identifier.doi10.1002/pbc.28354en
dc.identifier.urihttp://hdl.handle.net/10541/623013
dc.description.abstractLetter to editoren
dc.language.isoenen
dc.relation.urlhttps://dx.doi.org/10.1002/pbc.28354en
dc.titleGenomic profiling of acute myeloid leukaemia associated with ataxia telangiectasia identifies a complex karyotype with wild-type TP53 and mutant KRAS, G3BP1 and IL7Ren
dc.typeArticleen
dc.contributor.departmentAcademic Department of Medical Genetics, Addenbrooke's Hospital, University of Cambridge, Cambridge, UK.en
dc.identifier.journalPediatric Blood and Canceren
dc.description.noteen]
refterms.dateFOA2020-06-16T12:25:41Z


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