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dc.contributor.authorJoshi SR
dc.contributor.authorKitagawa A, Jacob C
dc.contributor.authorHashimoto R
dc.contributor.authorDhagia V
dc.contributor.authorRamesh A
dc.contributor.authorZheng C
dc.contributor.authorZhang H
dc.contributor.authorJordan, Allan M
dc.contributor.authorWaddell, Ian D
dc.contributor.authorLeopold J
dc.contributor.authorHu CJ
dc.contributor.authorMcMurtry IF
dc.contributor.authorD'Alessandro A
dc.contributor.authorStenmark KR
dc.contributor.authorGupte SA
dc.date.accessioned2020-04-22T07:25:11Z
dc.date.available2020-04-22T07:25:11Z
dc.date.issued2020en
dc.identifier.citationJoshi SR, Kitagawa A, Jacob C, Hashimoto R, Dhagia V, Ramesh A, et al. Hypoxic activation of glucose-6-phosphate dehydrogenase controls the expression of genes involved in the pathogenesis of pulmonary hypertension through the regulation of DNA methylation. Am J Physiol Lung Cell Mol Physiol. 2020;318:L773-L86.en
dc.identifier.pmid32159369en
dc.identifier.doi10.1152/ajplung.00001.2020en
dc.identifier.urihttp://hdl.handle.net/10541/622929
dc.language.isoenen
dc.relation.urlhttps://dx.doi.org/10.1152/ajplung.00001.2020en
dc.titleHypoxic activation of glucose-6-phosphate dehydrogenase controls the expression of genes involved in the pathogenesis of pulmonary hypertension through the regulation of DNA methylationen
dc.typeArticleen
dc.contributor.departmentDepartment of Pharmacology, New York Medical College, Valhalla, New York.en
dc.identifier.journalAmerican Journal of Physiology Lung Cellular and Molecular Physiologyen
dc.description.noteen]


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