Show simple item record

dc.contributor.authorMatsushita, Men
dc.contributor.authorIwasaki, Ten
dc.contributor.authorNonaka, Daisukeen
dc.contributor.authorKuwamoto, Sen
dc.contributor.authorNagata, Ken
dc.contributor.authorKato, Men
dc.contributor.authorKitamura, Yen
dc.contributor.authorHayashi, Ken
dc.date.accessioned2017-10-23T19:34:19Z
dc.date.available2017-10-23T19:34:19Z
dc.date.issued2017-09
dc.identifier.citationHigher expression of activation-induced cytidine deaminase is significantly associated with Merkel cell polyomavirus-negative Merkel cell carcinomas. 2017, 60 (3):145-153 Yonago Acta Meden
dc.identifier.issn0513-5710
dc.identifier.pmid28959124
dc.identifier.urihttp://hdl.handle.net/10541/620616
dc.description.abstractMerkel cell carcinomas (MCCs), clinically aggressive neuroendocrine skin cancers, are divided into Merkel cell polyomavirus (MCPyV)-positive and -negative tumors, which show different clinicopathological features and may develop through different mechanisms of carcinogenesis. Aberrant expression of activation-induced cytidine deaminase (AID) as a genomic modulator was demonstrated through pathogen-related NF-κB signal in Helicobacter pylori-associated gastric cancer, adult T cell leukemia/lymphoma (HTLV-1), hepatoma (HCV), and Burkitt lymphoma (EBV).
dc.language.isoenen
dc.rightsArchived with thanks to Yonago acta medicaen
dc.titleHigher expression of activation-induced cytidine deaminase is significantly associated with Merkel cell polyomavirus-negative Merkel cell carcinomas.en
dc.typeArticleen
dc.contributor.departmentDepartment of Pathobiological Science and Technology, School of Health Science, Tottori University Faculty of Medicine, Yonago 683-8503, Japanen
dc.identifier.journalYonago Acta Medicaen
html.description.abstractMerkel cell carcinomas (MCCs), clinically aggressive neuroendocrine skin cancers, are divided into Merkel cell polyomavirus (MCPyV)-positive and -negative tumors, which show different clinicopathological features and may develop through different mechanisms of carcinogenesis. Aberrant expression of activation-induced cytidine deaminase (AID) as a genomic modulator was demonstrated through pathogen-related NF-κB signal in Helicobacter pylori-associated gastric cancer, adult T cell leukemia/lymphoma (HTLV-1), hepatoma (HCV), and Burkitt lymphoma (EBV).


This item appears in the following Collection(s)

Show simple item record