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dc.contributor.authorValassi, E
dc.contributor.authorCrespo, I
dc.contributor.authorKeevil, B
dc.contributor.authorAulinas, A
dc.contributor.authorUrgell, E
dc.contributor.authorSantos, A
dc.contributor.authorTrainer, Peter J
dc.contributor.authorWebb, S
dc.date.accessioned2017-01-13T10:24:05Z
dc.date.available2017-01-13T10:24:05Z
dc.date.issued2017-02
dc.identifier.citationAffective alterations in patients with Cushing's syndrome in remission are associated with decreased BDNF and cortisone levels. 2017, 176 (2):221-231 Eur J Endocrinolen
dc.identifier.issn1479-683X
dc.identifier.pmid27932530
dc.identifier.doi10.1530/EJE-16-0779
dc.identifier.urihttp://hdl.handle.net/10541/620078
dc.description.abstractAffective alterations and poorer quality of life often persist in patients with Cushing's syndrome (CS) in remission. Brain-derived neurotrophic factor (BDNF) regulates the hypothalamic-pituitary-adrenal axis (HPA) and is highly expressed in brain areas controlling mood and response to stress. Our aims were to assess affective alterations after long-term remission of CS and evaluate whether they are associated with serum BDNF, salivary cortisol (SalF) and/or cortisone (SalE) concentrations.
dc.language.isoenen
dc.rightsArchived with thanks to European journal of endocrinologyen
dc.titleAffective alterations in patients with Cushing's syndrome in remission are associated with decreased BDNF and cortisone levels.en
dc.typeArticleen
dc.contributor.departmentEndocrinology/Medicine DepartmentHospital Sant Pau, Centro de Investigacion Biomedica en Red de Enfermedades Raras (CIBERER, Unidad 747), IIB-Sant Pau, ISCIII and Universitat Autonoma de Barcelona (UAB), Barcelonaen
dc.identifier.journalEuropean Journal of Endocrinologyen
refterms.dateFOA2020-04-21T08:22:30Z
html.description.abstractAffective alterations and poorer quality of life often persist in patients with Cushing's syndrome (CS) in remission. Brain-derived neurotrophic factor (BDNF) regulates the hypothalamic-pituitary-adrenal axis (HPA) and is highly expressed in brain areas controlling mood and response to stress. Our aims were to assess affective alterations after long-term remission of CS and evaluate whether they are associated with serum BDNF, salivary cortisol (SalF) and/or cortisone (SalE) concentrations.


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