Show simple item record

dc.contributor.authorHendry, Jolyon H
dc.date.accessioned2010-06-11T14:29:15Z
dc.date.available2010-06-11T14:29:15Z
dc.date.issued1991-10
dc.identifier.citationThe slower cellular recovery after higher-LET irradiations, including neutrons, focuses on the quality of DNA breaks. 1991, 128 (1 Suppl):S111-3 Radiat. Res.en
dc.identifier.issn0033-7587
dc.identifier.pmid1924735
dc.identifier.urihttp://hdl.handle.net/10541/104729
dc.description.abstractData for many biological end points reviewed here indicate that the responsible lesions are not only more severe but also are often more slowly repaired as the LET of the ionizing radiation is increased. This is reflected in high RBE values for end points assayed at long (in contrast to short) times after irradiation, for example, residual injury in normal tissues and carcinogenesis.
dc.language.isoenen
dc.subject.meshAnimals
dc.subject.meshDNA
dc.subject.meshDNA Damage
dc.subject.meshDNA Repair
dc.subject.meshDNA, Single-Stranded
dc.subject.meshEnergy Transfer
dc.subject.meshNeutrons
dc.subject.meshRadiation Injuries, Experimental
dc.titleThe slower cellular recovery after higher-LET irradiations, including neutrons, focuses on the quality of DNA breaks.en
dc.typeArticleen
dc.contributor.departmentCancer Research Campaign Department of Radiation Biology, Paterson Institute for Cancer Research, Christie Hospital, Manchester, United Kingdom.en
dc.identifier.journalRadiation researchen
html.description.abstractData for many biological end points reviewed here indicate that the responsible lesions are not only more severe but also are often more slowly repaired as the LET of the ionizing radiation is increased. This is reflected in high RBE values for end points assayed at long (in contrast to short) times after irradiation, for example, residual injury in normal tissues and carcinogenesis.


This item appears in the following Collection(s)

Show simple item record