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The PI3K-AKT-mTOR axis persists as a therapeutic dependency in KRAS<SUP>G12D</SUP>-driven non-small cell lung cancer

McDaid, William J
; 
Wilson, Leah
; 
Adderley, Helen
; 
Martinez-Lopez, A.
; 
Baker, Martin J
; 
Searle, Joshua
; 
Ginn, Lucy
; 
Budden, Timothy
; 
Aldea, M.
; 
Marinello, A.
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Abstract
Recommendations are based on available scientific data and the authors’ collective expert opinion.
Authors
McDaid, William J
Wilson, Leah
Adderley, Helen
Martinez-Lopez, A.
Baker, Martin J
Searle, Joshua
Ginn, Lucy
Budden, Timothy
Aldea, M.
Marinello, A.
Aredo, J. V.
Viros, Amaya
Besse, B.
Wakelee, H. A.
Blackhall, Fiona
Castillo-Lluva, S.
Lindsay, Colin R
Malliri, Angeliki
Affiliation
Division of Cancer Sciences, School of Medical Sciences, Faculty of Biology Medicine and Health, The University of Manchester, Manchester, UK; Cell Signalling Group, Skin Cancer and Ageing Group, Cancer Research UK Manchester Institute, The University of Manchester, Manchester, UK; Cancer Research UK Lung Cancer Centre of Excellence, The University of Manchester, Manchester, UK; The Christie NHS Foundation Trust, Manchester, UK;
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Date
2024
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All Christie Publications
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Article
Citation
McDaid WJ, Wilson L, Adderley H, Martinez-Lopez A, Baker MJ, Searle J, et al. The PI3K-AKT-mTOR axis persists as a therapeutic dependency in KRAS<SUP>G12D</SUP>-driven non-small cell lung cancer. MOLECULAR CANCER. 2024 NOV 12;23(1). PubMed PMID: WOS:001353603800001. English.
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http://hdl.handle.net/10541/627409
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